Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the other hand, our capacity to rescue SERT operate in KI midbrain synaptoneurosomes through the inhibition of FAK indicates elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in five-HT https://pro3390000.blog2news.com/33004586/getting-my-pro33-login-to-work